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From within cells, ROS can disrupt pathways including the small GTPase RhoA, PI3, and protein kinase B (PKB/Akt) signaling pathways

We hypothesized that high 15LO1, PEBP1, and GPX4 activity drives abnormal asthmatic redox biology, evidenced by lower bronchoalveolar lavage (BAL) fluid and intraepithelial cell GSH:oxidized GSH (GSSG) ratios, to enhance type 2 (T2) inflammatory responses