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Lipoic acid was found to effectively slow disease progression when administered either orally (87), intraperitoneally (88), or subcutaneously (89) to mice with experimental autoimmune encephalomyelitis (EAE), a model of multiple sclerosis

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Previously, it has been reported that acute exposure to GLP-1 did not significantly affect the metabolome 40 , nor bioenergetics of rodent -cells 15

hGPR17L regulation of GLP-1 secretion was Gq-independent and dependent upon Gi/o signaling, but was not correlated with MDL29,951-induced whole-cell cAMP signaling

The Xaa7 to Xaa38 corresponds to amino acids substitutions in those positions, the U being a spacer, and B is an acidic terminal group