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186,187,509,510 The accumulated DNA oxidative damage in AD hyper-activates the DNA damage sensor PARP-1, thereby reducing the concentration of cellular NAD + and suppressing the function of NAD + -SIRT1-PGC-1 axis, which in turn causes abnormal mitochondria
Current understanding relies heavily on extrapolation from consistent animal model results and accumulating anecdotal human reports
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Selenium is also an essential component of other metabolic systems that deal with oxidative stress, such as the thioredoxin system
doi: 10.1016/j.ebiom.2016.06.017 82
The number of glutathione injections varies by individual needs
