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The cancer metabolic reprogramming and immune response

The complexity of redox signaling in neoplasms underscores its potential as both a pathogenic hallmark and a therapeutic vulnerability

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Chemically defined conditions for human iPSC derivation and culture

Similarly, HsaGST A2-2, which has minimal ketosteroid isomerase activity was mutated in five H-site residues to attain a 3,500-fold higher catalytic efficiency with 5-AD (Pettersson et al., 2002)

MOTS-c is an exercise-induced mitochondrial-encoded regulator of age-dependent physical decline and muscle homeostasis