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In practice, that same additive quality is also the main risk: two strong satiety agents together can suppress appetite and slow the gut more than intended, which is why tolerability not maximum weight effect tends to be the limiting factor in any amylin-plus-incretin discussion
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This also prolongs the reliance on anaerobic sources of ATP: relative hyperglycolysis after TBI has been observed in the absence of hypoxia, when PbtO 2 is normal [70]
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