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Adems, en todos estos experimentos se demuestra que los efectos citoprotectores de GLP-1 (7-36) estn principalmente mediados por mecanismos dependientes de la activacin de la va RISK, principalmente de la fosfoinositol 3-quinasa (PI3K) y de las quinasas reguladas por seales extracelulares (ERK1/2) (fig
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