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This Cop1 is responsible for driving TAM recruitment, M2 polarization, and promoting resistance to immunotherapy and its inhibition leads to enhanced antitumor responses to PD-1 blockade (156)

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They also reported that this compound blocks CaV2.3 (R-type) and CaV2.2 (N-type) VGCCs and decreases excitation in the spinal card, as shown in Figure 4C (Shan et al., 2019)

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P-values from correlation analyses were adjusted for multiple comparisons using the BenjaminiHochberg false discovery rate (FDR) procedure at q = 0.05, applied separately within each group

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