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A mechanistic model of ADHD as resulting from dopamine phasic/tonic imbalance during reinforcement learning

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The misplaced mtDNA pathologically hyperactivated the cGAS-STING innate immune sensing pathway, culminating in an explosive downstream cascade of type I interferons and sterile inflammation (67)

The neurorestorative effects appear mediated through BDNF elevation, synaptic remodeling, and potentially neurogenesis in specific brain regions