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Selenium deficiency induces duodenal villi cell apoptosis via an oxidative stress-induced mitochondrial apoptosis pathway and an inflammatory signaling-induced death receptor pathway
The benefits depend on composition, standardization, bioavailability and the capacity to activate key cellular pathways
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During sepsis, cytokine storms can lead to the overexpression of nitric oxide (NO) synthase on endothelial cells and the synthesis of NO, resulting in vasodilation and adrenergic hyporeactivity, which affects microcirculatory hemodynamics (135)
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