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Strategic peptide combinations work through complementary mechanisms rather than overlapping ones
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Heterogeneous ferroptosis susceptibility of macrophages caused by focal iron overload exacerbates rheumatoid arthritis
A hypothesized mechanism is attributed to L-cells that co-produce GLP-1 and GLP-2 at the same time, which regenerates intestinal epithelium, mediates peptide YY production, and subsequently, through appetite suppression, prolongs satiety through GLP-1 receptors and thus manages weight control [33, 47]
These cells coordinate the complex interplay between keratinocytes and inflammatory cells by secreting growth factors and inflammatory mediators, thereby contributing to the regulation of epithelial homeostasis
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