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& Wright, J
& Bernard, D
Sikiric, P., Jadrijevic, S., Seiwerth, S., et al
Benchmarking in vitro tissue-engineered blood-brain barrier models
They also report that after 48 hours the rate of GSH transport out of the cell declined by 38%
Considering the physiological role of selenoproteins in antioxidation and the detrimental effects of oxidative stress in chondrocytes, aberrant selenium metabolism is likely to disrupt cartilage homeostasis and cause arthropathic diseases via dysregulation of redox homeostasis (Fig
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