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Even when fertilization is achieved, oxidative stress (inflammation)-induced apoptosis (cell death) may result in embryo fragmentation implantation failure insufficient luteal hormonal levels miscarriage impaired placenta development inadequate growth of the uterine lining (necessary for embryo development congenital abnormalities More reproductive condition-specific
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While its primary action involves p53 release, the modulation of FOXO4-related pathways intersects with cellular stress-response systems
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