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Its pharmacology is relatively narrow: it stimulates lipolysis through beta-3 adrenergic receptor pathways in adipose tissue and may inhibit lipogenesis [1]

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On the other hand, overexpression of Nrf2 (transcriptional regulator of seleno-GPxs) contributed to the increase in the radioresistance and stemness in glioma stem cells (GSCs) by upregulating GPx1 and decreasing the cellular ROS level

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