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We hypothesized that even among patients who developed acute pancreatitis, GLP-1 [RAs] could be safely continued

Lipid peroxidation and the subsequent cell death transmitting from ferroptotic cells to neighboring cells

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However, like most therapies targeting metabolic pathways, they are not free of risks

However, further investigation is needed to understand how these DEGs and pathways translate into the broader systemic effects that become evident following the engagement of CNS GIPR and GLP-1R