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Figure 5: Changes in tissue weight in lean mice and obese mice across the three different experimental treatment groups

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Overall, current data substantiates a strong mechanistic rationale and an early clinical signal for natural compounds in ASD, especially in subgroups with oxidative or mitochondrial dysfunction or nutritional deficits

Archived from the original on 6 May 2019

Refusal to share this information or other critical safety details should raise concerns