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Therefore, cortisol may prevent rather than cause oxidative stress and may activate antioxidant defences via genomic pathways in addition to influencing other systems that regulate the formation of pro-oxidants like ROS (65)

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Due to neurons having a limited capacity for GSH synthesis, they must rely on astrocytes to supply precursors such as cysteine (Prez-Sala D and Pajares, 2023)

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Several sources of ROS have been identified during diabetes, including enhanced blood glucose flux through the polyol and hexosamine pathway, increased formation of advanced glycation end products, and activation of protein kinase C pathway (137, 138)

Such a system-level property may link molecular reactions to tissue-scale dynamics