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The altered levels of aldosterone likely explain the hypokalemia and metabolic alkalosis presenting with Gitelman syndrome

This concern was related to the action of DPP-4 to cleave a number of bioactive peptides with alanine or proline as the second amino acid from the N-terminal end apart from GLP-1 and GIP, such as neuropeptide Y, gastrin-releasing peptide, substance P, and various chemokines (25, 26)

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Inhibition of pyruvate dehydrogenase kinase 4 ameliorates kidney ischemia-reperfusion injury by reducing succinate accumulation during ischemia and preserving mitochondrial function during reperfusion