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By selectively disrupting key antioxidant systemssuch as inhibiting glutathione synthesis with buthionine sulfoximine (BSO), blocking thioredoxin reductase with auranofin, or directly targeting NRF2therapies can push cancer cells beyond their limited oxidative buffering capacity

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HK-2 cells that had undergone various treatments were fixed in 4% paraformaldehyde

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However, the non- or slow biodegradability of pristine MSNs and MONs can lead to long-term retention in vivo, thus inducing possible toxicity risks