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Amino acid mutations in the H-site can significantly alter the catalytical activity of GST enzymes towards their substrates [88, 89]

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Mediation of IGF-1-induced skeletal myotube hypertrophy by PI(3)K/Akt/mTOR and PI(3)K/Akt/GSK3 pathways

The 33-mer DGP fits perfectly with HLA-DQ2 or HLA-DQ8 and triggers an immune response, leading to inflammation and small intestine villous atrophy

The same mechanism that extends the replicative life of healthy cells also supports the proliferation of cancer cells, which are defined by their ability to bypass normal telomere-based growth limits

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