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Further supporting this, Lock and colleagues demonstrated that glutathione protects against 2-chloropropionic acid (CPA)-induced cerebellar toxicity by preserving thiol redox equilibrium and potentially interacting with NMDA receptor-mediated pathways
10.1038/sj.bjp.0704191
Vi nhng kh nng ny, Glutathione khng ch n thun l mt sn phm lm trng m cn l mt dng cht cn thit cho ln da khe mnh
Glutamatergic modulators: the future of treating mood disorders
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