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Similar concerns apply to phosphodiesterase-5 inhibitors (medications like sildenafil) and L-arginine supplements that also influence nitric oxide signaling

Second, the EEG endpoint that defined the compounds activity is a population-level pattern in the recipient cortex, not a molecular readout meaning the discovery established a phenomenology, not a pathway

the proceedings note that these summarised points were not formally endorsed by the National Academies [14]