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These findings suggest that while FOXO4 is involved in the senescence pathway, it also plays a role in maintaining the viability of senescent Leydig cells by suppressing their apoptotic response, which could contribute to the accumulation of senescent cells [1]

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Chrousos et al

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Research from large-scale gene profiling studies suggests that GHK-Cu may regulate the activity of over 4,000 human genes reactivating those involved in tissue repair and regeneration while downregulating those associated with inflammatory and destructive processes