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In vivo studies with long-acting glucagon analogue suggest that hypoaminoacidaemia could be an important contributing mechanism to the observed increase in energy expenditure with glucagon agonism, but it also leads to enhanced lean mass loss [63]
Curr Opin Chem Biol 81:102496 Funding No funding was obtained for this study
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Li T, Chandrashekar A, Beig A, Walker J, Hong JKY, Benet A, et al
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