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CO can selectively impede the functioning of TNF-, IL-1 and macrophage inflammatory proteins, which are pro-inflammatory cytokines, triggered by LPS [57, 58]

doi: 10.1038/s41392-024-01931-z

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It operates in conjunction with GIP (Glucose-dependent Insulinotropic Polypeptide) to elicit a 2threefold augmented insulin response