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FOXO4-DRI, a novel peptide-based intervention, specifically disrupts the interaction between FOXO4 and P53, thereby inducing apoptosis in senescent cells

Impairments in the ETC can lead to electron leakage and the excessive generation of reactive oxygen species (ROS)

While supplementation with exogenous GSH has shown therapeutic potential on its own, recent findings indicate advantages to combined treatment with other immunomodulatory or anti-infective agents (Vairetti et al., 2021)

Biomed Pharmacother = Biomed Pharmacother (2020) 130:110519

96 However, GSH supplementation might protect pathogens by scavenging ROS, reducing drug efficacy in certain contexts