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Thus, the phenotypes of PAR2 mutant mice with differential protease sensitivity indicate a critical role for selective protease activation of PAR2 in regulating metabolism in DIO
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Once the oxidative fire is put out, the mitochondria can begin the slow process of repairing their damaged structural components
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These considerations suggest that GLP-1RA biology is best interpreted within a broader metabolic context in which substrate availability, redox buffering capacity, and tissue-specific energetic constraints shape systemic adaptation to sustained pharmacological weight reduction
Rangaraju V, Calloway N, Ryan TA
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