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revealed that endogenous metabolites of vitamin E, particularly -13-COOH, exert anti-inflammatory effects by directly inhibiting 5-lipoxygenase (5-LO), a key enzyme in leukotriene biosynthesis

When SAM is reduced from MTHFR-impaired methionine regeneration, EZH2 and other histone methyltransferases lose substrate availability producing progressive loss of repressive histone methylation marks at senescence-promoting gene promoters

NAC helps to refill glutathione levels in the body

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Cyclooxygenases and the prostaglandins they form also have roles beyond inflammation