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Supplementing with creatine monohydrate has been shown to increase muscle phosphocreatine stores, supporting ATP regeneration during high-intensity exercise and leading to greater strength, power, and muscle mass gains

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Nevertheless, another study stated that the interaction between POR and its downstream electron receptors was not required for ferroptosis induction, and POR was sufficient to independently impair PUFA-containing phospholipid membranes[29]

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Furthermore, even after the initiation of insulin therapy, uncertainty remains regarding which additional antihyperglycaemic agents should be introduced and at what stage, especially in the context of long-term GCs treatment