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Following cleavage by caspase-1 or caspase-4/5/11, the GSDMD N-terminal fragment forms pores in the tumor cell membrane, leading to osmotic imbalance, cell lysis, and the release of tumor-associated antigens [86]

At the same time, resistance to leptin during obesity impairs the regulation of hunger and energy use, further contributing to fat accumulation [30,31,32]

CPPs can form complex with semaglutide and significantly enhance the delivery across nasal epithelium cells through endocytosis process

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