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Current gene therapy research aims to target, for example, upstream targets of the GSH system or molecules associated with it (such as Nrf2, GPX, SLC7A11), genes aberrantly expressed in myocardial injury, and noncoding RNAs regulating mRNA transcription [76, 202, 250,251,252,253]

This increase in Tregs accelerated MASH, as evidenced by the fact that depleting Tregs dramatically inhibited MASH progression [76]