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Maintaining adequate hydration before and after administration may support normal metabolic and detoxification processes during therapy

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The use of MET inhibitors may disrupt typical downstream pathways, specifically JAK/STAT1, which are common to both MET and IFN-, thereby impeding the IFN--triggered expression of PD-L1 and PD-L2 in tumors with MET amplification

GlycoATP and mitoATP were derived from cells sequentially treated with oligomycin A and Rot/Antimycin A

Blood flow to the skin increases by up to 30%