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This dysfunction results in excessive influx of extracellular Ca and concurrent release of calcium from intracellular stores such as the endoplasmic reticulum and sarcoplasmic reticulum, culminating in a pathological elevation of cytosolic Ca concentrationa hallmark of calcium overload [1]

He et al

H., Luzia L

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Modulated human maternal and premature neonatal erythrocyte membrane enzyme activities in relation to the mode of delivery: in vitro restoration with L-carnitine

Serine: 750mg 13