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In both in vitro (0.5 M for 24 h) and in vivo (141 mol/kg/day for 14 days) settings, SF mitigated high glucose-induced apoptosis by inhibiting the upregulation of apoptotic markers and the downregulation of antiapoptotic maker Bcl-2 protein in hippocampal neurons exposed to high glucose (126)
These symptoms typically diminish over 48 weeks as tolerance develops
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