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Effects of environmental factors on APAP-induced mortality The above data demonstrate that our Drosophila -based system recapitulates studies performed in mice and demonstrates a similar pathologic mechanism for acetaminophen toxicity in Drosophila

Excess iron resulted in ROS formation through the Fenton reaction (14, 15)

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Therefore, inhibiting the MVA pathway can downregulate the synthesis of selenocysteine tRNA, thereby affecting GPX4 activity and inducing ferroptosis

TBIs can further be classified as penetrating and closed-head injuries