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The study showed that the drug interacted with the GLP-1 receptor to a much higher degree than with the glucagon receptor, indicating that any pharmacological effects were likely mediated via GLP-1R activation

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sealants/adhesives (4-tert-amylphenol,4-tert-butylphenol), lubricating oils, disinfectants and natural substances such as the blackwood dust used in making furniture (Knight 2008)

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