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Short-chain acyl-CoA ( ~ 5 carbons in the aliphatic tail) diffuses passively across the mitochondrial membrane
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GSH augmentation deserves investigation as a beneficial therapeutic approach for not only PD but also numerous other CNS disorders for which GSH deficiency and GSH-related enzyme deficits have been documented (multiple sclerosis, 25,26 autism, 2729 Alzheimers disease, 30,31 schizophrenia, 32,33 and bipolar disease 34 )
(February 2011)
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