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Promotes tendon-to-bone healing and collagen organization in rat models at levels comparable to or exceeding the bodys natural regenerative processes after injury
Cell Cycle 11 , 21592169 (2012)
Mechanistic pathways identified in animal and in vitro work include: FAK-paxillin pathway activation: BPC-157 stimulates focal adhesion kinase (FAK) and its downstream effectors, promoting fibroblast migration and tendon cell outgrowth
Its strength lies in its ability to accelerate tendon and ligament recovery, a.k.a
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