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suggest that the pathogenesis of mitochondrial-associated epilepsy involves processes such as neuronal energy depletion, oxidative stress, impaired calcium signaling, neurotransmitter imbalances, -oxidation deficiency, and glial cell dysfunction (3)
8-oxo-dG is one of the major products of DNA oxidation
Capsules: 120
doi: 10.3390/cells13030248 196 ZengLYangKYuGHaoWZhuXGeAet al
Commu 10 , 111 (2019)
The randomized, double-blind, placebo-controlled trials were conducted over a period of 48 weeks and involved numerous intramuscular doses provided over several weeks and long-term safety monitoring was performed until week 74
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