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These findings correspond well with earlier observations using lower doses of the two peptides [13], and demonstrate an additive effect

Amylin acts primarily on the brainstem and hypothalamusareas of the brain that regulate hunger, satiety, and energy balance

This is the window where the medication begins to interact with the hunger centers in the hypothalamus

The medication works by mimicking the incretin hormone GLP-1, which stimulates insulin secretion, suppresses glucagon release, and slows gastric emptying