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Using a Flexstation 3 plate reader, a baseline luminescence reading was acquired for 8 minutes at 37C, followed by stimulation with vehicle or 100 nM exendin-4 for Nb37 bystander assay, or with serial dilutions of exendin-4 starting from 1 M, 100 nM, 10 nM to 1 nM for 30 minutes for the mini-G s / mini-G q / -arrestin 2 recruitment assays
179,180 Deficiency or point mutation of chromodomain helicase DNA-binding protein 7 (CHD7), a chromatin remodeler with adenosine triphosphatase (ATPase) activity, is the causative factor of CHARGE syndrome
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49: 463468, doi: Ringseis R, Keller J, Eder K
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