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During the antigen recognition process, ROS are generated which function as second messengers to activate inflammatory signaling networks like MAPK and NF-B, and GSH buffering maintains the conditions to support those pathways (Schieber and Chandel, 2014)

This concept has been the ideal for many years, but with the limited number of viable islets and the need for immunosuppression, it has been limited to a very few, and the outcomes have not always been favourable

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Figure 1: Changes in A) escape latency and B) time spent in quadrants across all treatment groups

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