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Front Med 8(9):1060581 Acknowledgements Thanks to Fondazione Roma for the continuous support to our scientific research

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BPC-157s primary signalling mechanisms are well-characterised: it activates VEGFR2, drives downstream ERK1/2 phosphorylation, and stimulates nitric oxide synthesis via the Akt-eNOS axis producing pro-angiogenic, fibroblast-activating, and extracellular matrix-stabilising effects in experimental models

If you have already tried and failed first-line therapies or advanced biologics, subbing in an unverified peptide exposes you to a genuine risk of an uncontrolled disease flare