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GSNO was used solely as a nitrosylating agent to generate S-nitrosylated TTR, as previously shown [64], and excess reagent was removed prior to analysis

Both compounds require refrigerated storage after reconstitution

10.1210/en.2009-1123 Endocrinology 54 LermanG.AviviC.MardoukhC.BarzilaiA.TessoneA.GradusB.et al (2011)

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Int J Biol Sci Mitochondrial SIRT2-mediated CPT2 deacetylation prevents diabetic cardiomyopathy by impeding cardiac fatty acid oxidation