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Sudakov KV, Umriukhin PE, Koplik EV, Anokhin KV

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226 Aberrant sialylation can promote CD4 + T-cell activation, leading to the occurrence of UC, while deficiency of the -2,6-sialyltransferase ST6GAL1 inhibits the process of sialylation and limits CD4 + T cell activation by suppressing the expression of NF-B, subsequently decreasing the production of proinflammatory cytokines and alleviating UC

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A study conducted using a rat model of type 1 diabetes demonstrated reduced inflammatory cell infiltration and decreased glomerular expression of inflammatory markers, including TGF-1 and intercellular adhesion molecule-1, with administration of exendin-4 [51]

The effect of BPC 157 on muscle function is combined with the counteraction of increased levels of pro-inflammatory and pro-cachectic cytokines and of downstream pathways to abolish muscle cachexia [2]