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M., Alam, U

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Clinically, strategies aimed at modulating EAAC1-mediated cysteine transport and boosting endogenous GSH synthesis may hold promise for mitigating post-ischemic neurodegeneration and cognitive deficits associated with vascular dementia

Regression and remission were poorly defined, methodologically inconvenient, and philosophically discordant with a field built around inevitable, irreversible eGFR loss