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The NF-B pathway functions as a central pro-survival and pro-inflammatory transcription factor that promotes tumor progression by suppressing tumor suppressors such as phosphatase and tensin homolog (PTEN), thereby indirectly enhancing PI3K/AKT signaling and supporting cancer cell viability

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However, cysteine has been shown to accumulate in tissues of patients with glutathione synthetase deficiency at least in cultured fibroblasts [24]

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