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Lo CWH, Fei Y, Cheung BMY
Caloric deficits increase whole-body EAA requirements, meaning the body needs more of these muscle-building amino acids at the exact time that appetite suppression makes them harder to consume
To further dissect the molecular mechanism underlying GLUD1 degradation via the proteasomal pathway, we sought to identify the mediator(s) that govern the ubiquitin-dependent degradation of GLUD1 under conditions where GLUD1 methylation is abrogated
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The SURMOUNT-5 data showing 47% greater weight loss with tirzepatide suggests that many partial semaglutide responders would cross the threshold into meaningful weight loss with the stronger medication
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