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Ablative surgery tries to alleviate these symptoms by creating a lesion within the internal globus pallidus, thalamus, or subthalamic nucleus to stop the overactive pathway either near the start, as in pallidotomies, or further down the line in thalamotomies [Figure 1] [74]

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However, these tumors show increased activation of key pathways that promote and inhibit ferroptosis, suggesting that targeting drugs that promote ferroptosis could be an effective clinical target

Interestingly, even infectious entities with little to no CNS penetrance can stimulate inflammatory responses within the brain parenchyma, suggesting a role for systemic inflammation in the etiopathogenesis of neuroinflammation

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