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The anti-apoptotic functions of GIP are largely mediated by the PKA-dependent phosphorylation of CREB, whilst the influence of GLP-1 on apoptosis requires PI3K [101, 104]

However, recent studies have highlighted that prorenin and/or active renin can bind to prorenin receptor (PRR), which is expressed in glomerular mesangial cells, collecting ducts, and the subendothelium of renal arteries