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In contrast to the Kataoka's mice model where PLC was expressed in a shortened and catalytically inactive form, the knockout models described by Martins and coauthors either have a complete loss of the PLC expression (PLC -/- ) or have expression of the mutant form of PLC with mutant RA domains enable to bind to Ras (PLC RAm/RAm )

10.1007/s40261-015-0368-5 11 AndrabiS

Though the exact mechanism is unclear, GIP levels can become raised without inducing hypoglycemia in normoglycemic patients [4]

It has been confirmed that IL-6 and its receptor led to chondrocyte ferroptosis by inducing cellular oxidative stress and interfering with iron homeostasis, which can be inhibited by overexpression of miR-10a-5p, and these findings suggest that the IL-6/miR-10a-5p/IL-6R axis could be a potential therapeutic target for intervention in IVDD (114)

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